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Glycogen Storage Disease (GSD)

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Version vom 17. Januar 2026, 01:31 Uhr von MaudeKpy66981943 (Diskussion | Beiträge) (Die Seite wurde neu angelegt: „<br>The electrical signal units off a series of events that lead to crossbridge cycling between myosin and actin, which generates pressure. The sequence of occasions is barely totally different between skeletal, clean and cardiac muscle. This hole known as the synapse. The neurotransmitter crosses the hole, binds to a protein (receptor) on the muscle-cell membrane and causes an action potential in the muscle cell. The motion potential quickly spreads alon…“)
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The electrical signal units off a series of events that lead to crossbridge cycling between myosin and actin, which generates pressure. The sequence of occasions is barely totally different between skeletal, clean and cardiac muscle. This hole known as the synapse. The neurotransmitter crosses the hole, binds to a protein (receptor) on the muscle-cell membrane and causes an action potential in the muscle cell. The motion potential quickly spreads along the muscle cell and enters the cell through the T-tubule. The motion potential opens gates within the muscle's calcium retailer (sarcoplasmic reticulum). Calcium ions movement into the cytoplasm, which is the place the actin and myosin filaments are. Calcium ions bind to troponin-tropomyosin molecules positioned within the grooves of the actin filaments. Normally, the rod-like tropomyosin molecule covers the sites on actin the place myosin can kind crossbridges. Upon binding calcium ions, troponin adjustments form and slides tropomyosin out of the groove, exposing the actin-myosin binding websites. Myosin interacts with actin by cycling crossbridges, as described beforehand.

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